Brain Edema / Cerebello Pontine Angle Tumors: by I. Klatzo (auth.), K. Schürmann, M. Brock, H.-J. Reulen, D.

By I. Klatzo (auth.), K. Schürmann, M. Brock, H.-J. Reulen, D. Voth (eds.)

This quantity includes the papers awarded on the twenty fourth Annual assembly of the Deutsche Gesellschaft fur Neurochirurgie, held in Mainz, Western Germany, on April 30 - might three, 1973. planned selection was once made up of the most important nonetheless hotly debated matters which, for a long time, have intended a resource of continuing fear, and nights with no sleep to each neurosurgeon. simply so long as our exact box exists, there were the issues of ways to manage mind edema and ·of tips to lessen lethality and the secondary lesions in surgical procedure of cerebello-pontine perspective tumors. about the first topic, new pathological, pathophysiological and chemical features, the mechanisms of mind edema formation and backbone are offered within the wish for higher figuring out. in addition, the connection among mind edema, intracranial strain, cerebral blood move and metabolism are mentioned. eventually, the therapeutical results in addition to the result of experimental and scientific paintings are awarded, and a comparability of results among diverse equipment (hypertonic ideas, diuretics, steroids, managed hyperventilation, hyperbaric oxygen) is given. about the moment major topic, any vital contributions to the early prognosis of cerebello-pontine perspective tumors were integrated. however, it's of maximum curiosity for the neurosurgeon to understand which procedure he's to desire for different phases of tumor measurement and to be accustomed to the trans­ labyrinthine process or the posterior craniotomy, in addition to with the significance of using the microscope in neurosurgery, the upkeep of the facial nerve and, on occasion, its repair.

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Extra resources for Brain Edema / Cerebello Pontine Angle Tumors: Pathophysiology and Therapy / Diagnosis and Surgery

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Severe brain edema after cerebral ischemia or anoxia develops in two phases. 21 First intracranial hypertension is confined to the supratentorial space. Because of the "Axialverschiebung" (RIESSNER, ZULCH, 1939) hypertension in the infratentorial space may follow after an interval of some hours. This is proved by the fact that the no-reflow phenomenon was observed in the infratentorial space later than in the supratentorial space. The well-known fact that in brain death a stop of the intracranial circulation is first observed in the carotid artery and later in the vertebral artery is another support for this assumption.

Severe brain edema after cerebral ischemia or anoxia develops in two phases. 21 First intracranial hypertension is confined to the supratentorial space. Because of the "Axialverschiebung" (RIESSNER, ZULCH, 1939) hypertension in the infratentorial space may follow after an interval of some hours. This is proved by the fact that the no-reflow phenomenon was observed in the infratentorial space later than in the supratentorial space. The well-known fact that in brain death a stop of the intracranial circulation is first observed in the carotid artery and later in the vertebral artery is another support for this assumption.

KLINTWORTH, G. K. : Evaluation of the role of neurosurgical procedures in the pathogenesis of secondary brain- stern hemorrhages. J. , Psychiatr. 29, 423-425 (1966) 10. , J. CERVOS-NAVARRO and H. SCHNEIDER: Experimental brain death. I. Morphology and fine structure of the brain. , Psychiatr. (in press) 11. ren Hirn5dems. Neurochir. 22, 129-166 (1970) 22 12. RIESSNER, D. and K. J. ZULCH: Uber die Formveranderungen des Hirns (Massenverschiebungen, Zisternenverquellungen) bei raumbeengenden Prozessen.

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