Coronary Artery Disease by James T. Willerson, David R. Holmes Jr.

By James T. Willerson, David R. Holmes Jr.

Cardiovascular medication: Coronary Artery sickness will provide ultra-modern latest, basic suggestions at the overview, prognosis, and scientific and surgery of this most crucial point of heart problems and should be at the want record for each trainee and practicising heart specialist, cardiac health practitioner, vascular health care provider, diabetologist, cardiac radiologist and any health care provider who manages cardiac sufferers. The e-book is devoted to supplying finished assurance of each point of coronary arterial medication from cardiac signs via imaging and the genetic foundation for coronary sickness to surgical procedure, interventions, therapy and likewise to preventive cardiology. The Editor – and a bunch of contributing international experts – provide their many years of clinical and medical adventure. a huge promoting element is the constant bankruptcy association, transparent layout, and fascinating textual content that incorporates uncomplicated positive factors akin to tables, lists and therapy packing containers. The reader can have the entire information to diagnose and deal with an entire variety of stipulations in a sequence of textbook assets, whereas additionally getting access to extra video fabric from the critical website.

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Buja LM, Willerson JT. The role of coronary artery lesions in ischemic heart disease: insights from recent clinicopathologic, coronary arteriographic, and experimental studies. Hum Pathol. 1987;18:451–61. 3. Buja LM, McAllister Jr HA. Coronary artery disease: pathological anatomy and pathogenesis. In: Willerson JT, Cohn JN, Wellens HJJ, Holmes Jr DR, editors. Cardiovascular medicine. 3rd ed. London: Springer; 2007. p. 593–610. 4. Buja LM, McAllister Jr HA. Atherosclerosis: pathologic anatomy and pathogenesis.

88. Clubb Jr FJ, Darrouzet SD, Roberts AW, Weeks BR, Buja LM. Integrated microscopy techniques for analyzing postmortem intravascular stents. Modern Pathol. 2011;24:74A. 89. Buja LM. Vascular responses to percutaneous coronary intervention with bare-metal stents and drug-eluting stents: a perspective based on insights from pathological and clinical studies. J Am Coll Cardiol. 2011;15:1323–6. 90. Willerson JT, Yao SK, McNatt J, Benedict CR, Anderson HV, Golino P, Murphree SS, Buja LM. Frequency and severity of cyclic flow alteration and platelet aggregation predict the severity of neointimal proliferation following experimental coronary stenosis and endothelial injury.

Reprinted with permission from Wolters Kluwer) stenosis and relieve myocardial ischemic symptoms [82–84]. Percutaneous transluminal coronary angioplasty (PTCA) can produce a variety of acute effects, including dilatation of the vessel caused by stretching of the intima and media, damage to the endothelial surface, multiple fissures in the plaque, and dissection of the media (Figs. 28) [85–89]. The acute injury initiates a reparative response which leads to intimal proliferation [85–89]. Similar effects occur after atherectomy and laser angioplasty.

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